Facial nerve palsy refers to weakness of the facial muscles on one side of the face (a partial loss of movement), whereas paralysis means a complete absence of movement. This condition develops when the facial nerve (cranial nerve VII) is damaged or its function is impaired.
The most common form is idiopathic facial paralysis, known as Bell’s palsy. It develops suddenly, usually without an identifiable cause. This type of paralysis is peripheral, meaning that the nerve itself is affected after exiting the brainstem.
The causes of facial nerve palsy are usually classified as primary (idiopathic) or secondary.
Symptoms typically appear suddenly and progress within a few hours. Facial asymmetry soon becomes evident: the nasolabial fold flattens, the corner of the mouth droops, and the patient is unable to wrinkle the forehead, raise the eyebrow, fully close the eye (lagophthalmos), or puff out the cheek. In addition, taste disturbances and increased sensitivity to sound (hyperacusis) may occur.
Diagnosis is primarily based on clinical examination. Particular attention should be given to excluding secondary causes, especially stroke. Treatment of Bell’s palsy is most effective when started within the first 72 hours and usually includes corticosteroids to reduce nerve swelling. Eye protection and lubrication are essential to prevent corneal damage.
It is important to distinguish between peripheral facial palsy, such as Bell’s palsy, and central facial palsy, which may result from stroke. In peripheral palsy, the entire side of the face, including the forehead muscles, is affected. By contrast, central palsy partially preserves forehead movement because of bilateral cortical innervation, allowing the patient to still wrinkle the forehead.
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